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E. Floyd Robinson, MD; Henry B. Soloway, MD

AMA Arch Surg. 1971;102(5):501-502.

Abstract
Acid-base and arterial oxygen pressure (Po₂) determinations were performed upon groups of rabbits with (1) fat embolism, (2) fat embolism plus hemorrhagic shock, and (3) hemorrhagic shock alone. The data suggest that the initial severe metabolic acidosis observed with fat embolism plus shock is principally a reflection of hypovolemia, and that the fat embolism per se contributes little to the acid-base derangements. Transient cyanosis with hypoxia was observed seven minutes following fat embolism, but this reverted by 30 minutes after embolization. In the group which received fat embolism plus shock, the mean 30 minute Po₂ was actually slightly higher than the comparable preembolism value.

Author Affiliations
Temple, Tex; Las Vegas, Nev

From the Department of Neurosurgery, Scott-White Clinic, Temple, Tex (Dr. Robinson), and the Department of Pathology, Southern Nevada Memorial Hospital, Las Vegas, Nev (Dr. Soloway).

Footnotes
Accepted for publication Dec 21, 1970.

Reprint requests to Southern Nevada Memorial Hospital, 1800 W Charleston Blvd, Las Vegas, Nev 89102 (Dr. Soloway).

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