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Carlton J. Young, MD
Birmingham, Ala

Arch Surg. 2002;137:605.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Laparoscopic donor nephrectomy has supplanted ODN as the procedure of choice in suitable patients. Hospitalization is shorter, pain management is easier, and return to work comes faster.¹ Historically, open donors have had good long-term renal function.² Because LDN is in its infancy, long-term results are not available, but the assumption based on the ODN data is that the results will be comparable.³

Laparoscopic donor nephrectomy is radically different from ODN. Pneumoperitoneum alters the donor's physiology significantly^4-5; therefore, the effect on the kidney could be profound. Hazebroek and colleagues seek to address one physiologic perturbation: the liberation of ADH in response to pneumoperitoneum.

Because ADH release follows the stimulation of extrarenal baroreceptors, ADH release in LDN is probably not due to pneumoperitoneum. The release of this hormone may be a secondary response of endothelin 1 (ET-1), which is produced and released from vascular endothelial cells as . . . [Full Text of this Article]

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Antidiuretic Hormone Release During Laparoscopic Donor Nephrectomy
Eric J. Hazebroek, Robert de Vos tot Nederveen Cappel, Diederik Gommers, Teun van Gelder, Willem Weimar, Ewout W. Steyerberg, H. Jaap Bonjer, and Jan N. M. IJzermans
Arch Surg. 2002;137(5):600-605.
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