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ParathyroidectomyTreatment For Secondary Hyperparathyroidism and Azotemic Osteodystrophy
James J. Berens, MD;
Daniel J. Potter, MD;
George M. Nickas, MD
AMA Arch Surg. 1970;100(4):338-342.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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The exciting developments in the understanding and management of osteodystrophy, due to primary hyperparathyroidism, are being recapitulated. We are now attacking the osteodystrophies produced by secondary hyperparathyroidism, which is due to chronic renal failure. Although the osseous changes of chronic renal failure have been observed for decades, the eventual terminal fatal course of chronic renal failure patients has precluded any surgical approach. Significant advances in recent years in nephrology have, in selected patients, reversed the inevitable downhill course of chronic renal failure by regular dialysis. These therapeutic accomplishments have not only allowed long-term survival in these particular patients but have also allowed the osseous changes to progress into a state of clinical significance and invalidism in patients whose general health has otherwise been good. Our purpose in this study will be to deal in particular with chronic renal failure patients whose osseous changes are induced by a state of secondary
. . . [Full Text PDF of this Article]
Author Affiliations
Phoenix, Ariz
From the departments of surgery (Dr. Berens) and nephrology (Drs. Potter and Nickas), Good Samaritan Hospital, Phoenix, Ariz.
Footnotes
Accepted for publication Dec 10, 1969.
Read before the 77th annual meeting of the Western Surgical Association, Dallas, Nov 20, 1969.
Reprint requests to 550 W Thomas Road, Phoenix, Ariz 85013 (Dr. Berens).
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