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Garland D. Perdue, MD; Robert B. Smith, III, MD; Leeon Rhodes, MD; William D. Long, MD

AMA Arch Surg. 1970;100(5):562-564.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Cerebral ischemia is caused by occlusive arterial disease anywhere along the course of the blood supply to the brain. Irreversible damage is most likely to occur when there is inadequate collateral circulation due to (a) involvement of multiple vessels; (b) inadequacy of anastomoses of the vascular structures at the base of the brain due to congenital variation or occlusive disease; or (c) when an artery terminal to the circle of Willis becomes occluded. The fact that cerebral infarction may result from disease in the surgically accessible portion of the cervical carotid artery is well known. The Joint Study of Extracranial Arterial Occlusion reports that 75% of 3,788 patients had accessible lesions, often combined with inaccessible lesions, but existing alone in 41%.¹ It is equally well known that cerebral infarction is usually preceded by premonitory symptoms of transient neurologic handicaps or minor strokes heralding the catastrophic event. These symptoms may . . . [Full Text PDF of this Article]

Author Affiliations
Atlanta

From the Joseph B. Whitehead Department of Surgery, Emory University School of Medicine, Atlanta.

Footnotes
Accepted for publication Nov 5, 1969.

Reprint requests to Emory University Clinic, Atlanta 30322 (Dr. Perdue).

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