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FURTHER STUDIES

W. E. ADAMS, M.D.; H. M. LIVINGSTONE, M.D.

Arch Surg. 1931;23(3):500-512.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In a recent publication¹ on the subject of obstructive pulmonary atelectasis, two etiologic factors were described as being essential to its production, viz., bronchial obstruction and labored respiration or expiration against resistance. The latter of these two factors was contrary to the observations of Lee,² Coryllos and Birnbaum³ and others,⁴ who had produced the condition experimentally with regularity, and who believed a quiet, shallow respiratory cycle to be one of the important factors in its etiology. This idea has been carried down from the time of Pasteur⁵ who, observing several cases of massive atelectasis associated with postdiphtheritic paralysis of the diaphragm, believed it due to a reduction in the depth of respiration.

When an attempt was made to reproduce the results of other investigators, the factor of straining respiration was accidently encountered and massive atelectasis produced with routine regularity in its presence.

That straining respiration . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the Department of Surgery, University of Chicago.

Footnotes
Submitted for publication, Nov. 21, 1930.

This work has been conducted under a grant from the Douglas Smith Foundation for Medical Research of the University of Chicago.

A preliminary report of this study was published in Proc. Soc. Exper. Biol. & Med. 27:982, 1930.

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