This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

EXPERIMENTAL AND CLINICAL STUDIES

JAMES C. WHITE, M.D.; WALTER EATON GARREY, M.D.; JAMES A. ATKINS, M.D.

Arch Surg. 1933;26(5):765-786.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The late Sir James Mackenzie is reputed to have said that discussions on angina pectoris are usually futile, as they simply consist of the replacement of one speculative hypothesis by another. This has been true both as to the etiology of the disease and as to the most logical surgical method of preventing the attacks. It is not within the scope of this paper to review the multitudinous theories which have been put forward to explain the cause of these painful crises; suffice it to say that by their very number Mackenzie's dictum has remained substantiated for many years. Recently, however, an increasing number of cardiologists have subscribed to the theory of Keefer and Resnik,¹ that most cases of angina pectoris can be accounted for on the basis of anoxemia of the myocardium. The experiments of Sutton and Lueth² have given strong support to this theory of the . . . [Full Text PDF of this Article]

Author Affiliations
BOSTON

From the Surgical Services of the Massachusetts General Hospital.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?