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LESTER R. DRAGSTEDT, Ph.D., M.D.; H. E. HAYMOND, M.D.; JAMES C. ELLIS, M.D.

Arch Surg. 1934;28(2):232-291.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Introduction

Etiology of Acute Pancreatic Necrosis

Common Channel Theory and Objections to It

Relation of Bile to Acute Pancreatic Necrosis

Nonbiliary Causes of Acute Pancreatic Necrosis

Mechanism of Production of Acute Pancreatic Necrosis

Does Intraglandular Activation of Trypsin Cause Acute Pancreatic Necrosis?

Mechanism of Production of Acute Pancreatic Necrosis by Bile

Summary

Cause of Death in Acute Pancreatic Necrosis

Observations on the Toxicity of Pancreatic Juice

Intraperitoneal Injection of Fresh, Actively Proteolytic Pancreatic Juice

Intravenous Injection of Fresh, Actively Proteolytic Pancreatic Juice

Intraperitoneal Injection of Fresh, Actively Proteolytic Pancreatic Juice, Sterilized by Berkefeld Filtration

Intraperitoneal Injection of Fresh, Actively Proteolytic Pancreatic Juice from the Dog into Mice

Comment

Observations on the Toxicity of the End-Products of Pancreatic Digestion and Autolysis and the Rôle of Bacteria in the Toxemia of Acute Pancreatic Necrosis

In Vivo Autolysis of Dog Pancreas Complicated by the Presence of Bacteria Commonly Found in the Uncontaminated Gland . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the Department of Surgery of the University of Chicago.

Footnotes
This work has been aided by a grant from the Douglas Smith Foundation for Medical Research of the University of Chicago.

Preliminary reports were made at the meeting of the American Physiological Society in Chicago, March 27, 1930; at the meeting of the Chicago Society of Internal Medicine, May 15, 1930, and at the meeting of the Chicago Surgical Society, March 6, 1931.

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