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A POSTMORTEM STUDY

DOUGLAS E. SCOTT, M.B.

Arch Surg. 1934;28(2):296-306.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

That dilatation of the ureter will result from obstruction is apparent, but that it may be acquired in the absence of any form of mechanical obstruction is perhaps less obvious and at first glance paradoxical. Atony of the ureter, by which is meant loss of functional ability of the muscle of the ureter, whereby peristalsis is weak or absent, was first recognized by Russian observers as a cause of ureteral dilatation. Further, they conceived the idea that atony and dilatation may result from inflammation. In this country Braasch repeatedly has called attention to this inflammatory dilatation, but by most observers such a possibility has not been appreciated or has been frankly doubted.

It is also true that certain obstructive factors commonly assumed to cause ureteral dilatation are in fact of obscure mechanism. Thus, there has been much speculation over the ureteral dilatation which accompanies pregnancy, and also over that which . . . [Full Text PDF of this Article]

Author Affiliations
GREENWICH, CONN.

Footnotes
Abstract of thesis submitted to the Faculty of the Graduate School of the University of Minnesota in partial fulfilment of the requirements for the degree of Master of Science in Urology, May, 1931. The work was done while the author was a Fellow in Urology, the Mayo Foundation, Rochester, Minn.

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