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NORMAN W. ROOME, M.D.; PAUL E. McMASTER, M.D.

Arch Surg. 1934;29(1):54-58.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The question of the influence of circulatory changes on the healing of bone has been discussed by many authors with conflicting opinions. Pearse and Morton¹ and McMaster and Roome² have offered experimental evidence that the healing of defects of bones is accelerated by venous stasis. On the other hand, sympathectomy, with its increased flow of blood in the extremity, has been empirically advocated in the treatment of fractures by Leriche,³ Colp and Mage⁴ and others. Pearse and Morton,⁵ Key and Moore⁶ and McMaster and Roome² found no experimental confirmation of the value of such therapy. Clinically, certain atrophies of bone are explainable on the basis of arterial hyperemia (Greig⁷). Harris⁸ listed several conditions known to cause hypertrophy and lengthening of the bones of children in which various degrees of venous stasis exist.

Formation of bone may be initiated by the transplantation . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO; LOS ANGELES

From the Department of Surgery, the University of Chicago.

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