This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

GENE H. KISTLER, M.D.

Arch Surg. 1934;29(4):589-611.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

A number of lesions, etiologically obscure but with certain characteristics in common, have been described in various bones. The onset and clinical progress are insidious and may or may not be associated with trauma. In these lesions there is a gradual and progressive destruction of the tissues of the bone, as demonstrated in roentgen films and by examination of tissue. The parts involved are almost always in an epiphysis or in epiphyseal cartilage, and ultimately a deformity of the bone results. These various lesions have been designated osteochondritis, anemic infarction, epiphysitis or aseptic necrosis of bone, terms which imply some vascular or nonspecific disorder not clearly understood. Among the more important specifically named disturbances of this group are: aseptic or anemic necrosis of the head of the femur, radius or humerus or distal epiphysis of the femur; König's osteochondritis dissecans of the medial condyle of the femur; osteochondritis deformans juvenilis . . . [Full Text PDF of this Article]

Author Affiliations
UNIVERSITY, ALA.

From the Henry Baird Favill Laboratory of St. Luke's Hospital, Chicago, John Jay Borland Fellowship for Clinical Research, and the Department of Physiology and Pharmacology, University of Alabama Medical School.

Footnotes
Awarded the Joseph A. Capps Prize for Medical Research, 1933.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?