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GABRIEL P. SELEY, M.D.

Arch Surg. 1939;39(5):783-791.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Numerous attempts have been made to produce bacterial peritonitis in animals. In some experiments attempts were made to enhance the pathogenic effect of bacteria by preliminary treatment of the peritoneum of laboratory animals with a great variety of substances: turpentine (David¹), agar (David and Loring²), croton oil (Pawlowsky³), potato (Halsted⁴), omental ligation (Halsted⁴), sarcolin (Solieri⁵), intestinal juices (Blalock⁶) and tragacanth (Benians;⁷ Steinberg⁸). In other experiments different inoculums were tried: feces (Herrmann⁹), fecal emulsions (Wilen and Dragstedt¹⁰) and contents of small intestines (Pawlowsky³). The intestinal tract has been employed as the source of peritoneal infection by ligation of the appendix (Costain;¹¹ Lehman¹²), division of the appendix (Blalock¹³), ligation of the mesoappendix (Blalock¹³), perforation or incision of the gastrointestinal tract at different levels (Bergh¹⁴) or drainage of an open loop of ileum into the peritoneal . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

Ralph Colp Fellow in Bacteriology.; From the Laboratories of the Mount Sinai Hospital

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