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C. FRANK CHUNN, M.D.; HENRY N. HARKINS, M.D., Ph.D.; R. T. BOALS, M.D.

Arch Surg. 1941;43(5):773-788.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Since 1933, when Sanguinetti¹ first called attention to increased amounts of urea nitrogen in the blood of patients with bleeding peptic ulcer, this observation has been confirmed by many writers. These include Sanguinetti himself² (1933), Christiansen³ (1935, 1936, 1937), Ingegno⁴ (1935), Meyler⁵ (1935, 1936), Sui⁶ (1935), Alsted⁷ (1936), Borst⁸ (1936, 1938), Clausen⁹ (1936) and Polack¹⁰ (1936). There have been at least sixteen other reports during the past five years; these will be quoted later. Although this syndrome has apparently been recognized over a period of only eight years, it is of considerable importance. The time has come to take stock and to appraise the significance, cause and mechanism of this syndrome.

SIGNIFICANCE OF AZOTEMIA

In some early studies the opinion was expressed that renal or hepatic damage was the cause of the elevation in urea nitrogen content of the blood . . . [Full Text PDF of this Article]

Author Affiliations
DETROIT

From the Department of Surgery, Henry Ford Hospital.

Footnotes
Read before the Section on Pathology and Physiology at the Ninety-Second Annual Session of the American Medical Association, Cleveland, June 6, 1941.

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