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EXPERIMENTAL AND CLINICAL EXPERIENCES WITH EUCUPINE AND PROCAINE HYDROCHLORIDE

REYNOLD PATZER, M.D.; VINCENT DERBES, M.D.; HUGO ENGELHARDT, M.D.

Arch Surg. 1945;50(6):296-300.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Clinicians have long suspected that migraine may be mediated through involvement of the cranial arteries. Many patients have discovered that pressure on the particular artery supplying the painful area will ameliorate the pain. Furthermore, Cushing¹ commented on the striking dilatation of the temporal arteries and veins seen during an attack of migraine. It remained for Wolff² and his co-workers to establish this relationship. In a series of experiments they were able to show that in many cases of migraine the fundamental difficulty was vascular dilatation. They demonstrated that changes in the intensity of headache are related to changes in the amplitude of pulsations in the cranial arteries, chiefly the branches of the external carotid, i. e., the superficial temporal and the occipital. Mechanical experimental distention of the superficial temporal artery by increasing the intravascular pressure produced pain of a migrainous type. It seems probable, however, that pial and . . . [Full Text PDF of this Article]

Author Affiliations
NEW ORLEANS

From the Departments of Surgery and Medicine, Tulane University of Louisiana School of Medicine and the Ochsner Clinic.

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