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An Experimental Study

PAUL C. COLONNA, M.D.; EVERETT D. JONES, M.D.

Arch Surg. 1948;56(2):161-171.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

THE CLINICAL recognition of aseptic necrosis of bone is based largely on the appearance of changes in the roentgenogram, and these of necessity must be the result of slowly developing factors on one of the resisting tissues of the body—bone. Death of bone tissue without evidence of infection has been described in various regions of the body in both childhood and adult life, and the factor or factors affecting its prevention or progress are of practical value. The bone infarct in the diaphysis may be asymptomatic, but whenever the pathologic process involves one of the articular surfaces, it will give rise to limitation in the joints and produce severe disability. In childhood Freiberg's disease, Perthes' disease and Köhler's disease are all representative of a localized aseptic necrosis. In the adult the development of aseptic necrosis in the head of the femur following a traumatic dislocation or a fracture of the . . . [Full Text PDF of this Article]

Author Affiliations
PHILADELPHIA

From the Department of Orthopaedic Surgery, University of Pennsylvania School of Medicine.

Footnotes
Read before the Section on Orthopedic Surgery at the Ninety-Sixth Annual Session of the American Medical Association, Atlantic City, N. J., June 13, 1947.

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