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REVASCULARIZATION FOLLOWING EXPERIMENTAL MESENTERIC VASCULAR OCCLUSION
RUDOLF J. NOER, M.D.;
JOHN WILLIAM DERR, M.D.
Arch Surg. 1949;58(5):576-589.
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THE SMALL intestine has a remarkable ability to withstand interruption of its blood supply. The distribution and anastomoses of its vessels appear to be responsible for its great revascularizing potential, and it has been generally assumed that the peculiar arcuate system of the intestinal tract is the chief factor in overcoming vascular deprivations. Considerable experimental effort has been devoted to determining the degree of vascular interruption which is compatible with life, yet the exact mode by which compensation takes place has not been clearly established. The literature pertinent to these studies may be discussed under two categories—(1) anatomic studies and (2) experimental mesenteric vascular interruption and revascularization of the intestine.
ANATOMIC STUDIES
Morphologic reports on the larger vessels of the small intestine are in accord, but many discrepancies appear in descriptions of the vasa recta and the vessels within the intestinal wall. Much of the confusion doubtless arises from the
. . . [Full Text PDF of this Article]
Author Affiliations
DETROIT
From the departments of surgery and anatomy, Wayne University College of Medicine.
Footnotes
Read at the Fifty-Sixth Annual Meeting of the Western Surgical Association, St. Louis, Dec. 2, 1948.
This investigation was supported by a research grant from the Division of Research Grants and Fellowships of the National Institute of Health, United States Public Health Service.
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