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SLUDGED BLOOD FOLLOWING SEVERE THERMAL BURNS
FRANKLYN BROOKS, M.D.;
LESTER R. DRAGSTEDT, Ph.D., M.D.;
LOUISE WARNER, Ph.D.;
MELVIN H. KNISELY, Ph.D.
Arch Surg. 1950;61(3):387-418.
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THE PURPOSE of this paper is to report the presence and to discuss the significance of sludged blood following severe thermal burns. Three sets of pathologic mechanisms are proposed: (1) a mechanism for the production of burn shock, (2) a mechanism for the production of postburn anemia and (3) a mechanism seemingly capable of being at least a factor in the postburn "toxemia."
GENERAL PROBLEMS
The pathologic alterations in the physiology of mammals following thermal burns have been attributed to almost as many factors as there have been investigators in the field. However, in spite of a great deal of accumulated experimental data, the factors determining the altered physiology of human patients during the first twenty-four to seventy-two hours following a severe burn continue to present important clinical problems. Prior to the introduction of tannic acid therapy by Davidson in 1925, the mortality rate within the first forty-eight hours after
. . . [Full Text PDF of this Article]
Author Affiliations
CHICAGO; BETHESDA, MD.; CHARLESTON, S. C.
From the Department of Surgery and the Hull Laboratory of Anatomy, University of Chicago, and the Department of Anatomy, Medical College of the State of South Carolina, Charleston, S. C.
Footnotes
This work was aided by grants from the Dr. Wallace C. and Clara A. Abbott Memorial Fund of the University of Chicago, from the Ella Sachs Plotz Fund.
The work described in this paper was done under a contract recommended by the Committee on Medical Research between the Office of Scientific Research and Development and the University of Chicago.
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