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ROBERT ELMAN, M.D.; T. E. WEICHSELBAUM, Ph.D.; Marjorie A. Graul, R.N.

AMA Arch Surg. 1951;62(5):683-697.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

EVEN though glycosuria and acetonuria are fairly common after operation, their exact significance is not generally known. Do they mean a serious disturbance in carbohydrate metabolism? Is the excretion of ketone bodies evidence of a transient overproduction thereof by the liver or of diminished utilization by peripheral tissue? Is the glycosuria the result of a diminution of peripheral utilization of glucose or of increased glycogenolysis? A number of surgical patients were observed in whom quantitative determinations of the 24 hour output of glucose and of acetone were made in an attempt to answer these questions. A few of these observations were briefly reported in a previous paper from this laboratory.¹

PREVIOUS WORK

In an exhaustive report published in 1933, Roscher² studied and reviewed postoperative acidosis and ketonuria. A disturbance in acid-base balance was common, particularly in patients operated on under general anesthesia. A fall in the pH and . . . [Full Text PDF of this Article]

Author Affiliations
ST. LOUIS

From the Washington University School of Medicine, Department of Surgery, and Barnes Hospital.

Footnotes
Aided by a grant from the Commonwealth Fund.

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