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A. BLAUSTEIN, M.D.; C. HUBER, M.D.; M. ALBERIAN, B.S.

AMA Arch Surg. 1951;63(5):592-598.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

COMPONENTS of the coagulation mechanism have been and are being extensively investigated in relation to the development of thrombosis. Most recently, Ochsner and his associates¹ have demonstrated the lowering of circulating anti-thrombin postoperatively, and they feel that this may play a role in the development of postoperative thrombosis. McFarlane² feels that trauma may cause extensive damage to tissues and blood vessels, with inevitable absorption of thromboplastin and thrombin. Ochsner¹ states that a predisposing factor to phlebothrombosis is the increased coagulability of the blood which occurs in persons subjected to tissue trauma. He further states that, whatever the cause, tissue damage results in blood changes which increase the coagulability of blood. Allen, Barker, and Hines³ observed that postoperative thrombophlebitis is commonest after procedures in which considerable tissue is removed and in which there is likely to be a great amount of tissue injury. Mason and Harrison⁴ . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

Dr. Blaustein is Chief of the Division of Hematology, Gouverneur Hospital, New York, and the Laboratory of the Bayonne Hospital, Bayonne, N. J.; Dr. Huber is Resident in Surgery, New York Polyclinic Medical School and Hospital, New York.

Footnotes
This study was supported by a grant from the Abbott Laboratories, North Chicago, Ill.

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