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  Vol. 75 No. 5, November 1957 TABLE OF CONTENTS
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  SURGERY IN ACUTE TRAUMA
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Mechanisms and Management of Cold Injury

CAPT. ELBERT B. FOUNTAIN, MC

AMA Arch Surg. 1957;75(5):716-717.

Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The pathologic changes resulting from freezing and nonfreezing cold injuries are identical, although generally more tissue is involved in the nonfreezing injuries, such as trench foot or immersion foot. In addition, maceration of the skin of the affected part is commonly observed in the nonfreezing entities. These are to be considered as caused by the effects of moisture, and not as the result of cold. The various tissues of the body do not show the same degree of susceptibility to cold injury. Nerves and striated muscle are highly susceptible; skin, fascia, and connective tissue are quite resistant, but not as resistant as compact bone or tendon. Blood vessels are highly susceptible to injury, resulting in leakage of plasma into the surrounding tissue. Necrosis as a rule does not occur in blood vessels as a result of cold. This relative tissue susceptibility has resulted in the observation of muscle gangrene without . . . [Full Text PDF of this Article]


Author Affiliations

U. S. Army


Footnotes

Submitted for publication May 23, 1957.

Read at the Tripler Army Hospital Symposium on Surgery in Acute Trauma, Honolulu, April 1-5, 1957.



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