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F. HENRY ELLIS, Jr., M.D.; ROBERT O. BRANDENBURG, M.D.; JOHN A. CALLAHAN, M.D.; HIRAM W. MARSHALL, M.D.

AMA Arch Surg. 1959;79(2):222-237.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Since the successful clinical introduction of extracorporeal circulation¹ in 1953, the majority of operations performed with the aid of this technique have been for congenital heart disease. Open heart surgery for acquired heart disease has developed more slowly, and mitral insufficiency, among other acquired valvular defects, only recently has come under direct surgical attack. Until relatively recently, pure mitral insufficiency was considered a rarity and of debatable significance with regard to cardiac function. We now know that mitral insufficiency, although less common than mitral stenosis as a predominant lesion, is a recognizable entity and can be a progressive disabling disease.

The anatomic factors which may underlie incompetence of the mitral valve are many and complex. They have been reviewed in some detail by Edwards and Burchell² and need not be repeated here. It is of importance, however, to recognize that "mitral insufficiency begets mitral insufficiency." That is to . . . [Full Text PDF of this Article]

Author Affiliations
Rochester, Minn.

Section of Surgery (Dr. Ellis); Section of Medicine (Drs. Brandenburg and Callahan), Mayo Clinic and Mayo Foundation. Fellow in Physiology (Dr. Marshall), Mayo Foundation. The Mayo Foundation. Rochester, Minn., is a part of the Graduate School of the University of Minnesota.

Footnotes
Submitted for publication Feb. 27, 1959.

Read at the 16th Annual Meeting of the Central Surgical Association, Montreal, Feb. 20, 1959.

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