This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on Web of Science (16)  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

JOSEPHUS C. LUKE, M.D., F.R.C.S. (Eng. and C.); B. A. LEVITAN, M.D., F.R.C.P. (C.)

AMA Arch Surg. 1959;79(2):269-275.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Impaired blood supply to one kidney as a cause of generalized arterial hypertension has been known for many years.^1,2 The classical experiments by Goldblatt³ in 1934 Placed this etiology on a firm foundation. Since then many nephrectomies have been Performed for this reason but with cure of the hypertension in only a disappointing minority because cases of renal parenchymal disease as well as those with renal arterial disease were being so treated. Thompson and Smithwick⁴ in 1952 found that their best results came when the origin of the renal ischemia was in the renal artery rather than in the kidney proper.

Although nephrectomy is an accepted method of therapy, the vasculature of the kidney removed in cases of renal artery stenosis or occlusion is relatively normal on pathological examination because it has been "protected" from the nephrosclerotic changes incident to the hypertension. The remaining kidney is unprotected. . . . [Full Text PDF of this Article]

Author Affiliations
Montreal

From the Medical and Surgical Services, Royal Victoria Hospital.

Footnotes
Submitted for publication Feb. 27, 1959.

Read at the 16th Annual Meeting of the Central Surgical Association, Montreal, Feb. 19, 1959.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?