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BRUCE R. HEINZEN, M.D.

AMA Arch Surg. 1960;81(4):627-631.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

This study was prompted by the current interest in the so-called ulcerogenic tumors of the pancreas.¹ Reported cases have led to the speculation that a hormone-like mechanism may arise in, or be closely related to, the non-β cells of the islets of Langerhans. A possibility that neural mechanisms, or neuroendocrine mechanisms, might be involved, seemed to require a restudy of the basic anatomy of the nerve endings in the pancreas.

Method

Human autopsy material, studied after staining with silver albuminate and pyridine in the presence of electrolytically active copper (technique of Bodian²), suggested that fixation artefacts interfered with the study of fine nerve fibers. Dog pancreas was then studied after fixation by perfusion of the vascular system at the time of death. Formolsaline is used, and further fixation in formalin by immersion is done. In fresh human autopsy material the celiac axis was perfused with fixative and the . . . [Full Text PDF of this Article]

Author Affiliations
Manhasset, N. Y.

Departments of Surgery, North Shore Hospital, Manhasset, Long Island and Cornell Surgical Division, Bellevue Hospital, Manhattan, New York.

Footnotes
Submitted for publication Feb. 3, 1960.

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