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CHARLES T. REYNOLDS, M.D.; SHELDON C. SOMMERS, M.D.

AMA Arch Surg. 1962;85(3):390-393.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Clinically an established glomerulonephritis with hypertension may be difficult to distinguish from other forms of renal disease.^1,2 In 9 of 1,380 patients who underwent bilateral lumbodorsal sympathectomy and renal biopsy in the treatment of hypertension during the years 1942 through 1958, a primary glomerulonephritis was not discovered preoperatively despite various chemical parameters of renal function, pyelography, and a careful history and physical examination. Repeated urinalyses failed to provide evidence indicative of glomerulonephritis, such as an increased excretion of protein, red cells, white cells, and granular casts. No patient had the nephrotic syndrome. In 3 of the cases, the clinical picture was that of a malignant, or nearly malignant, hypertension. This has been described as one of the masks of chronic glomerulonephritis.³ Histologically none were typical chronic proliferative glomerulonephritis, but instead were mild proliferative, membranous, inactive, or healed types.

The effect of sympathectomy upon this type of renal hypertension . . . [Full Text PDF of this Article]

Author Affiliations
BOSTON; LA JOLLA, CALIF.

From the Department of Surgery, Massachusetts Memorial Hospitals, Boston, and the Department of Pathology, Scripps Memorial Hospital, La Jolla, Calif.

Footnotes
Submitted for publication Oct. 28, 1961.

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