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MARTIN L. DALTON, JR., M.D.; JAMES D. HARDY, M.D.

AMA Arch Surg. 1963;86(3):455-459.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The problems encountered in the diagnosis and management of pulmonary arteriovenous fistulas are of much current interest. A1-though this lesion was apparently not recognized until 1897 and not successfully treated until 1942,¹ it has been reported with increasing frequency in recent years.^2-8 The relationship between pulmonary arteriovenous fistulas and hereditary hemorrhagic telangiectasia is well known.^9-11 The pulmonary lesions have also been found in association with Laennec's cirrhosis and thyroid carcinoma, although most pulmonary arteriovenous fistulas result from congenital vascular anomalies.¹²

The basic pathophysiologic lesion is a right-to-left shunt of varying magnitudes. The full-blown clinical syndrome consists of central cyanosis, clubbed fingers, and polycythemia in the presence of a normal heart, often with roentgenologic evidence of a mass lesion in the pulmonary parenchyma. An increase in total blood volume without a concomitant increase in plasma volume has been reported, although exact figures on this point are lacking. . . . [Full Text PDF of this Article]

Author Affiliations
JACKSON, MISS.

American College of Surgeons Mead-Johnson Scholar (Dr. Dalton).; Department of Surgery, University of Mississippi Medical Center.

Footnotes
Submitted for publication Oct. 2, 1962.

Aided by Army Contract No. DA-49-007-MD-627 and NIH Grant No. H-6163(C1).

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