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GUSTAVO BOUNOUS, MD; LAWRENCE G. HAMPSON, MD; FRASER N. GURD, MD

AMA Arch Surg. 1963;87(2):340-354.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The fundamental element in the treatment of hemorrhagic shock is the correction of the deficit of blood volume before critical changes have occurred which render the shock irreversible. In the dog which has been subjected to severe hemorrhagic shock the reinfusion of shed blood almost invariably leads to a recovery of blood pressure and cardiac output. This recovery is short-lived in animals which have passed the point of reversibility, and it is a matter of the greatest difficulty to explain why one animal will go on to survive while another will die.

During the period of hypotension the perfusion of tissues and the consumption of oxygen are both greatly reduced. The degree of depression of oxygen consumption during shock has been shown by Guyton and Crowell to bear a relationship to recovery.¹¹ Immediately after reinfusion the oxygen consumption rises along with the blood pressure and cardiac output, but it . . . [Full Text PDF of this Article]

Author Affiliations
MONTREAL

Fellow of the Royal College of Surgeons of Canada and Fellow of the American College of Surgeons (Dr. Hampson, Dr. Gurd); Medical Research Council (Canada) Scholar (Dr. Bounous).; From the McGill University Surgical Clinic of the Montreal General Hospital.

Footnotes
Presented at the 20th Annual Meeting of the Central Surgical Association, Chicago, Feb 21-23, 1963.

Assisted by grants-in-aid from the Medical Research Council and the Defense Research Board of Canada.

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