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Induced by Urea Infusions and Nephrectomy

RONALD FISHBEIN, MD; BENJAMIN F. RUSH, JR., MD; ROBERT J. WILDER, MD; GERALD P. MURPHY, MD

AMA Arch Surg. 1964;89(2):273-274.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In the course of metabolic studies in dogs following nephrectomy and ureteral ligation, it was noted that the chloride content of the excreta was often quite high. Since the excreta in this situation is almost entirely vomitus, we were attracted to a consideration of the components of gastric juice in uremia. The available data is sketchy in this regard. Peters¹ noted that the gastric aspirate in patients with chronic uremia was low in both chloride and titratable acidity. Schreiner and Maher³ in an extensive review of the subject of the gastrointestinal symptoms of uremia do not refer to any studies of the acidity or electrolyte content of gastric juice under these circumstances. They take note of the high incidence of uremic gastritis, heart burn, vomiting, and gastric bleeding in uremia, and are inclined to favor the high concentration of ammonia which develops in the intestinal tract as the . . . [Full Text PDF of this Article]

Author Affiliations
LEXINGTON, KY

From the departments of surgery of The Johns Hopkins Hospital, The Baltimore City Hospital, The Johns Hopkins University School of Medicine, and The University of Kentucky School of Medicine.

Footnotes
Submitted for publication Feb 6, 1964.

Supported by National Institutes of Health grant No. AM 06956-02 and H 5245 and Office of the Surgeon General contract No. DA-49-193-MD-2348.

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