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Diffuse Myocordial Infarction

JULIUS H. JACOBSON, II, MD; MICHAEL C. H. WANG, MD; TAKASHI YAMAKI, MD; HOWARD J. KLINE, MD; ALLAN E. KARK, MD; LESLIE A. KUHN, MD

AMA Arch Surg. 1964;89(5):905-914.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The present study was conducted to determine whether hyperbaric oxygenation provided protection to the heart in experimentally produced acute diffuse myocardial infarction. No attempt was made to simulate the entity of myocardial infarction as encountered clinically.

The use of high pressure environment to increase the delivery of oxygen to tissues is an old concept dating back to antiquity; the history, pressure, terminology, and physiology have been recently reviewed by Jacobson, et al.¹ Boerema^2,3 of Amsterdam and Illingworth et al⁴ of Glasgow have stimulated the current interest in its clnical application.

Hyperbaric oxygenation has been reported to be beneficial in a variety of ischemic situations, both clinical and experimental.^5-11 Smith and Lawson^10,12 ligated the circumflex branch of the left coronary artery in dogs, and reported protection with hyperbaric oxygenation against fibrillation and mortality. Meijne et al¹³ studied similar preparations, measuring coronary backflow, lactic acid, and . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Department of Surgery, Division of Vascular Surgery, and Department of Medicine, Division of Cardiology, Mount Sinai Hospital.

Footnotes
Read before the 12th Scientific Meeting of the International Cardiovascular Society, North American Chapter, San Francisco, June 20, 1964.

This work has been supported by USPHS grants HE-07706, HE-07707, and HE-04132.

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