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Experimental and Clinical Observations

James P. Carey, MD; Edward A. Stemmer, MD; John E. Connolly, MD

AMA Arch Surg. 1969;99(4):441-446.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Although the existence of abdominal angina as a clinical entity distinct from angina pectoris was proposed in the early 1900's,¹ it was not generally accepted even as late as 1931 that intraabdominal pain of vascular origin could occur in the absence of gangrene or peritonitis.² In 1936, Dunphy³ reported seven patients in whom recurrent attacks of abdominal pain preceded the development of fatal mesenteric occlusion and emphasized the importance of recognizing that transient abdominal pain could result from intestinal ischemia. Mikkelsen,⁴ in 1957, postulated that abdominal angina could be cured, and intestinal infarction prevented by reestablishing flow through the diseased mesenteric vessels. The advisability of surgical correction of these vascular lesions is now generally accepted.

Atherosclerosis is the most common cause of chronic ischemia to the alimentary tract.⁵ It is generally believed that as long as the occlusive process is a gradual one, two of . . . [Full Text PDF of this Article]

Author Affiliations
Irvine, Calif

From the Department of Surgery, University of California, Irvine, and the Veterans Administration Hospital, Long Beach, Calif.

Footnotes
Submitted for publication Feb 27, 1969.

Reprint requests to 5901 E Seventh St, Long Beach, Calif 90801 (Dr. Stemmer).

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